Trump again again bans THC…

I agree we should keep it from those under 21. Plenty of adults do not drive.
So only non driving adults should be able to use cannabis. There is also a risk of developing dependence and addiction, particularly with frequent use, and impacts on pregnancy, infant development. Should we also ban it in fertile women? We wouldn't want to hurt developing babies.
 
So only non driving adults should be able to use cannabis. There is also a risk of developing dependence and addiction, particularly with frequent use, and impacts on pregnancy, infant development. Should we also ban it in fertile women? We wouldn't want to hurt developing babies.
same with sugar.
 
There are people who abuse it, but not everyone who drinks it is an abuser of it.

Plenty of studies show moderate drinking can actually be helpful.

Makes sense to me because humans have evolved with fermented drink/food for millions of years. Things are not generally inherently bad/good for you, they are dependent on how adapted our species is to the things.

If fruit were not available to early humans, our digestive systems would not have developed the ease of digesting it.
I did not mean to belittle folks who have issues with booze. I was just making a stupid joke.
 
STOP BREAKING THE LAW.:cig:

The marijuana when I was a kid had 1.5 to 3 % THC the marijuana today is 15-20% and some gummies have 30% THC. Marijuana has many bad effects on humans. We don't need to make it even more accessible to kids. A recent study in Ohio showed 41.9% of deceased drivers in car crashes had THC in their systems.
Fast food has bad effects. So does alcohol, caffeine and sugar and trans fats.

Irrational argument
 
Really? Link?

Rada, Avena & Hoebel (2005): In rats with intermittent access to sucrose, daily bingeing repeatedly increases dopamine (DA) in the nucleus accumbens shell — a brain area central to reward and addiction. PubMed+1

Sucrose sham-feeding study: Even when stomach content is “sham fed” (i.e., not actually digested), just tasting sugar on a binge-schedule triggers accumbens dopamine release. PubMed

Kir2.1 Study (2017): Mice that had prolonged sucrose access, upon withdrawal, displayed depression- and anxiety-like behaviors. At the molecular level, there was an increase in the Kir2.1 potassium channel in the nucleus accumbens, and reduced dopamine/CREB activity. PubMed

Neuroimmune Effects (2024): Sex-specific effects in mice — after sucrose withdrawal, male mice showed neuroinflammatory marker increases in reward regions (PFC, NAc) and anxiety-like behavior. PubMed

Anxiety after deprivation (2008): Rats bingeing on sucrose then fasted showed anxiety and changes in extracellular acetylcholine and dopamine in the nucleus accumbens. PubMed

Deprivation-Effect Paradigm: Rats with intermittent sugar access show increased sugar-seeking behavior after abstinence, similar to “craving” in drug models. PubMed

Review of Sugar Addiction (Avena, Rada, Hoebel): A comprehensive animal model showing “bingeing,” “withdrawal,” “craving,” and “cross-sensitization” with drugs of abuse. PubMed


Added Sugar & Placental DNA Methylation (2021): Higher maternal added sugar intake was associated with altered DNA methylation in the placenta, as well as fetal behavior (reduced third-trimester movement) and shorter gestation. PubMed

Review of Sugar Use in Pregnancy: A review summarizing evidence that maternal sugar intake may contribute to gestational diabetes, preeclampsia, and may affect offspring metabolism, taste preference, and obesity risk. PubMe

DTI Study in Infants (2021): In human infants, higher prenatal maternal sugar intake (particularly “added sugar”) was associated with differences in brain tissue organization (via diffusion tensor imaging). The authors suggest this may reflect altered dendrite/synapse formation in gray matter. MDPI

Project Viva – Cognition Study (2018): In a cohort of ~1,234 mother-child dyads, higher maternal sucrose intake and sugar-sweetened beverage consumption during pregnancy was linked to lower cognitive scores in mid-childhood (non-verbal IQ), after adjusting for confounders. PubMed

High-Sugar Maternal Diet in Rats (2021): Maternal high-sugar intake during pregnancy/lactation impaired offspring memory, with molecular changes in NMDA receptor subunit composition in hippocampus and prefrontal cortex. PubMed

Prenatal Sucrose in Rats (Aged Offspring, 2017): High prenatal sucrose led to worse spatial memory in aged offspring and signs of oxidative stress + altered NMDA/Wnt signaling in hippocampus. PubMed

Epigenetic Mechanisms (Mice, 2024): A high-fat, high-sugar maternal diet in mice led to changes in offspring brain DNA-methylation enzymes (DNMT, TET) in a sex-specific way, suggesting a mechanistic pathway for neurodevelopmental risk. PubMed

Transgenerational Cognitive Deficits (Mice): Maternal over-nutrition (high fat + sugar) in mice caused cognitive deficits not only in the immediate offspring, but across multiple generations, with changes in neurotransmitter systems. PMC+1
 
Rada, Avena & Hoebel (2005): In rats with intermittent access to sucrose, daily bingeing repeatedly increases dopamine (DA) in the nucleus accumbens shell — a brain area central to reward and addiction. PubMed+1

Sucrose sham-feeding study: Even when stomach content is “sham fed” (i.e., not actually digested), just tasting sugar on a binge-schedule triggers accumbens dopamine release. PubMed

Kir2.1 Study (2017): Mice that had prolonged sucrose access, upon withdrawal, displayed depression- and anxiety-like behaviors. At the molecular level, there was an increase in the Kir2.1 potassium channel in the nucleus accumbens, and reduced dopamine/CREB activity. PubMed

Neuroimmune Effects (2024): Sex-specific effects in mice — after sucrose withdrawal, male mice showed neuroinflammatory marker increases in reward regions (PFC, NAc) and anxiety-like behavior. PubMed

Anxiety after deprivation (2008): Rats bingeing on sucrose then fasted showed anxiety and changes in extracellular acetylcholine and dopamine in the nucleus accumbens. PubMed

Deprivation-Effect Paradigm: Rats with intermittent sugar access show increased sugar-seeking behavior after abstinence, similar to “craving” in drug models. PubMed

Review of Sugar Addiction (Avena, Rada, Hoebel): A comprehensive animal model showing “bingeing,” “withdrawal,” “craving,” and “cross-sensitization” with drugs of abuse. PubMed


Added Sugar & Placental DNA Methylation (2021): Higher maternal added sugar intake was associated with altered DNA methylation in the placenta, as well as fetal behavior (reduced third-trimester movement) and shorter gestation. PubMed

Review of Sugar Use in Pregnancy: A review summarizing evidence that maternal sugar intake may contribute to gestational diabetes, preeclampsia, and may affect offspring metabolism, taste preference, and obesity risk. PubMe

DTI Study in Infants (2021): In human infants, higher prenatal maternal sugar intake (particularly “added sugar”) was associated with differences in brain tissue organization (via diffusion tensor imaging). The authors suggest this may reflect altered dendrite/synapse formation in gray matter. MDPI

Project Viva – Cognition Study (2018): In a cohort of ~1,234 mother-child dyads, higher maternal sucrose intake and sugar-sweetened beverage consumption during pregnancy was linked to lower cognitive scores in mid-childhood (non-verbal IQ), after adjusting for confounders. PubMed

High-Sugar Maternal Diet in Rats (2021): Maternal high-sugar intake during pregnancy/lactation impaired offspring memory, with molecular changes in NMDA receptor subunit composition in hippocampus and prefrontal cortex. PubMed

Prenatal Sucrose in Rats (Aged Offspring, 2017): High prenatal sucrose led to worse spatial memory in aged offspring and signs of oxidative stress + altered NMDA/Wnt signaling in hippocampus. PubMed

Epigenetic Mechanisms (Mice, 2024): A high-fat, high-sugar maternal diet in mice led to changes in offspring brain DNA-methylation enzymes (DNMT, TET) in a sex-specific way, suggesting a mechanistic pathway for neurodevelopmental risk. PubMed

Transgenerational Cognitive Deficits (Mice): Maternal over-nutrition (high fat + sugar) in mice caused cognitive deficits not only in the immediate offspring, but across multiple generations, with changes in neurotransmitter systems. PMC+1
I'm 100% against mice eating excessive amounts of sugar. They shouldn't use cannabis either even if most of them don't drive.
 
No they take it up the ass - like Jerry Fallwell Jr's wife from their pool boy - and that's WHILE Jerry Jr. sits in the corner and spanks it.

Think I'm joking? Think again, orange semen breath:


"Holy roller Jerry Falwell Jr.'s wife says she made sex tapes with pool boy"

You cannot be a true Christian if you support trump and what he does. You can only be a hypocrite. Which are you, @Stone ?
Unworthy of an actual response. Up to you to figure out why. Maybe you can start by asking yourself if its fair to accuse millions upon millions of Christians across the world of being deviants because of the actions of SOME.
 
So you support banning marijuana ?
The one problem I have with legalizing marijuana is that the active ingredient can remain in a person's body for up to 30 days. There currently are no laws regarding what a safe level of THC in someone is. Even if there were, that 30-day retention period is a serious problem.

So, if we still want to legalize it, and possibly some other 'recreational' drugs, I recommend the following:

For use of any legalized drug, you need to get a "Drug Abuser's License." That's what we should call it. It lists which drugs you can obtain and use. It is public information, and anyone can access the records to see who a licensed drug abuser is. If you don't have a license and get caught with those drugs, you go to prison for like 10 years on a first offense. That is, either you get the damn license or your life will be totally destroyed if caught doing drugs without it.

We license dealers and manufacturers in the same way.

Having a Drug Abuse License means a employer can, at will, fire you for having it without other cause. Having one makes you ineligible for things like a CDL, pilot's license, owning a firearm, etc. We don't want people doing those activities higher than a kite. Medical issues arising from your drug abuse are your problem. Insurers don't have to cover you for that.

I'd toss alcohol in with this too, if it had been invented yesterday, but it wasn't and there already are pretty stiff laws in place controlling it.

So, summing up, I'd legalize and stigmatize things like marijuana, etc., because simply legalizing it will cause more problems than it solves.
 
Unworthy of an actual response. Up to you to figure out why. Maybe you can start by asking yourself if its fair to accuse millions upon millions of Christians across the world of being deviants because of the actions of SOME.
Dude I could give a rat's ass about your perception of my worthiness. Get over yourself, princess 👸
 
Dude I could give a rat's ass about your perception of my worthiness. Get over yourself, princess 👸
Lie. If you didn't you wouldn't constantly stalk me practically begging for my attention. Its a little on the faggish side if you ask me. If I were you I'd cut back on the stalking some before people get the wrong impression of you and start thinking you are like your pal Diseasal.
 
Lie. If you didn't you wouldn't constantly stalk me practically begging for my attention. Its a little on the faggish side if you ask me. If I were you I'd cut back on the stalking some before people get the wrong impression of you and start thinking you are like your pal Diseasal.
LOL - yes. YOU set the standard for being manly? Were that the case - humanity would go extinct.

Speaking of fake Christians - how about the one who was boning porn stars and playmates during his marriage and who is currently trying to coverup an international pedophile ring - against the wishes of several Republicans?

You really need to find a new team to root for, Sally.

1763317566738.png
 
Back
Top