No, I was pointing out that you seem to think the shots protect the patient.
When a vaccine is 50% effective, the more vaccinated people you can surround patients with the better. Lets say that influenza has to travel through 3 vaccinated or unvaccinated people to get to a patient. If it is 3 vaccinated people, it has 1/8th the chance of getting to the patient as 3 unvaccinated people.
With contagious diseases, what other people do effects your health.
Son, there isn't a flu vaccine so right off the bat you look a fuckwit.
Wow, the things I learn from you are... "Interesting"? Is that the right word? Maybe "odd"?
Every year there is a mix of influenza vaccines against the probable strains of that year. I do not know why you believe it does not exist.
You don't know what a vaccine is do you?
The business should set up shots for the workers. Then they have no excuses. Wouldn't you feel better if you went to a place that all employees had shots?
Disagreed since Trump declared a medical emergency. Isn't that still in effect?
The Brazil strain of Covid-19 is definitely reinfecting people who are supposedly immune, and there is rising evidence that it is happening in America too. The post infection immunity appears to be weak, and fade quickly.
Post infection immunity only applies to a single strain of bug.
I know of no good [non-anecdotal] evidence of re-infection with Covid
I’d be willing to bet that instances of re-infection are due to the fact the first ‘infection’ was because the individuals received a false positive on their PCR test.
Sometimes that is correct, and other times that is not. It is far more complex than you are claiming here. Some diseases have different strains have very different entry mechanisms to the cell, and therefore have different immunities. So far there is no evidence of this in Covid-19. It appears that the newer strains are more infectious, and therefor more likely to get around weak natural immunities.
Anecdotal evidence is enough to prove it happens, so that is good enough. Anecdotal evidence cannot prove how often it happens. Studies out of the UK (not anecdotal) are beginning to show that reinfections mostly happen after 5 months. The British have been very good at sequencing the virus strains, and have better evidence than we do.
Brazil is showing definite reinfections, but they have less evidence of exactly what is happening, when.
How would the British sequence a false positive? If there is no virus, how would the sequence the RNA of the virus? You pre-lost this bet.
Before you can be *positive* of reinfection you have to be *certain* about the first infection. How are they doing this?
Finally, anecdotal evidence is prohibited.
no.Sometimes that is correct, and other times that is not. It is far more complex than you are claiming here. Some diseases have different strains have very different entry mechanisms to the cell, and therefore have different immunities. So far there is no evidence of this in Covid-19. It appears that the newer strains are more infectious, and therefor more likely to get around weak natural immunities.
no. the natural immunities (T cells and antibodies) would still be just as effective on the variants
same as any pathogen
Natural immunity fades over time. With influenza, it does not fade enough within a lifetime to matter much. With mononucleosis, it fades enough over 10 years that you can get the disease again. With Covid-19.... Who knows. It is too early to be sure.
The best guess is that it fades enough over 5 months. That is very quickly, but not impossible.
that wasnt at all what I was saying! But you at least get the drift. and dont forget the memory T and B cellsNatural immunity fades over time. With influenza, it does not fade enough within a lifetime to matter much. With mononucleosis, it fades enough over 10 years that you can get the disease again. With Covid-19.... Who knows. It is too early to be sure.
The best guess is that it fades enough over 5 months. That is very quickly, but not impossible.
When the British sequence the genetics of the virus, you can be sure the virus was there. That is just plain common sense. The virus is basically the genetics, and little more, so finding the full genetics says that the virus is there.
That is not even close to reality. Anecdotal evidence is of limited usefulness, but not prohibited. A good example is a statistically insignificant number of people have been to the moon. There is anecdotal evidence some people have been to the moon. Under your argument, the moon landing can be denied. Under my argument, it definitely happened, but we cannot know the odds of going to the moon.
that wasnt at all what I was saying! But you at least get the drift. and dont forget the memory T and B cells
that reignite long after the antibodies last.
Coronavirus: will immunity rapidly fade or last a lifetime?
March 5, 2021
https://theconversation.com/coronavirus-will-immunity-rapidly-fade-or-last-a-lifetime-155905
COVID-19 probably sits somewhere between measles and flu. It’s not as stable as measles and it is not as changeable as flu. We might expect immunity to last against COVID-19, but probably not as long as measles. And we’ll probably have to vaccinate against variants that emerge, as we do for flu.
One thing we’ve got going for us is the repetitive nature of the surface of SARS-CoV-2. The spike protein covers the surface of the coronavirus in a fairly uniform manner. Antibodies to smallpox, which also has a highly repetitive surface, last a lifetime. In this situation, macrophages (a type of white blood cell that engulfs and consumes pathogens) might be better able to gobble up the antibody-coated virus.
If the spike protein mutates and antibodies can’t bind as well, it’s well worth giving a booster shot against the new spike protein — which is what is planned.
not what it says at all. there are a ton of variablesYou own link says that immunity will likely fade quickly. Best guess is that you can really start getting Covid-19 again after an infection 5 months ago, but that is just a guess now.
And if antibodies don’t work as well against variants, T cells might. This might mean that we won’t need boosters at all and that we might have long-term protection against multiple variants. And even if the immune response is lower against variants, we will probably still be protected from severe disease.
The only way to determine if the virus ‘was there’ would be with an antibody test and even those are of limited usefulness with Covid.
It has nothing to do with sequencing since you can’t sequence a post-infection virus. That makes no sense.
There is plenty room for skepticism regarding Covid reinfection.
If it happens at all it won’t be a big driver of future Covid data.
what is "reinfection"? exposure? increased viral loading? symptomatic or asymptomatic?There is reasonable room for debate into the specifics of reinfection. How often does it happen? After what time frame?